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Pancreatic INS-1 β-Cell Response to Thapsigargin and Rotenone: A  Comparative Proteomics Analysis Uncovers Key Pathways of β-Cell Dysfunction  | Chemical Research in Toxicology
Pancreatic INS-1 β-Cell Response to Thapsigargin and Rotenone: A Comparative Proteomics Analysis Uncovers Key Pathways of β-Cell Dysfunction | Chemical Research in Toxicology

JNK activation by thapsigargin versus saturated free fatty acid (SFA).... |  Download Scientific Diagram
JNK activation by thapsigargin versus saturated free fatty acid (SFA).... | Download Scientific Diagram

Thapsigargin: key to new host-directed coronavirus antivirals?: Trends in  Pharmacological Sciences
Thapsigargin: key to new host-directed coronavirus antivirals?: Trends in Pharmacological Sciences

Effects of endoplasmic reticulum stress on apoptosis induction in  radioresistant macrophages
Effects of endoplasmic reticulum stress on apoptosis induction in radioresistant macrophages

Endoplasmic reticulum stress, the unfolded protein response and autophagy  in kidney diseases | Nature Reviews Nephrology
Endoplasmic reticulum stress, the unfolded protein response and autophagy in kidney diseases | Nature Reviews Nephrology

Frontiers | Mechanism of Endoplasmic Reticulum Stress in Cerebral Ischemia
Frontiers | Mechanism of Endoplasmic Reticulum Stress in Cerebral Ischemia

Targeting endoplasmic reticulum stress in insulin resistance: Trends in  Endocrinology & Metabolism
Targeting endoplasmic reticulum stress in insulin resistance: Trends in Endocrinology & Metabolism

Tunicamycin specifically aggravates ER stress and overcomes chemoresistance  in multidrug-resistant gastric cancer cells by inhibiting N-glycosylation |  Journal of Experimental & Clinical Cancer Research | Full Text
Tunicamycin specifically aggravates ER stress and overcomes chemoresistance in multidrug-resistant gastric cancer cells by inhibiting N-glycosylation | Journal of Experimental & Clinical Cancer Research | Full Text

Endoplasmic Reticulum Stress Induces G2 Cell-Cycle Arrest via mRNA  Translation of the p53 Isoform p53/47 - ScienceDirect
Endoplasmic Reticulum Stress Induces G2 Cell-Cycle Arrest via mRNA Translation of the p53 Isoform p53/47 - ScienceDirect

Thapsigargin promotes colorectal cancer cell migration through upregulation  of lncRNA MALAT1
Thapsigargin promotes colorectal cancer cell migration through upregulation of lncRNA MALAT1

Phenotypic Discovery of Neuroprotective Agents by Regulation of Tau  Proteostasis via Stress‐Responsive Activation of PERK Signaling - Shin -  2021 - Angewandte Chemie International Edition - Wiley Online Library
Phenotypic Discovery of Neuroprotective Agents by Regulation of Tau Proteostasis via Stress‐Responsive Activation of PERK Signaling - Shin - 2021 - Angewandte Chemie International Edition - Wiley Online Library

Calcium Influx Caused by ER Stress Inducers Enhances Oncolytic Adenovirus  Enadenotucirev Replication and Killing through PKCα Activation: Molecular  Therapy - Oncolytics
Calcium Influx Caused by ER Stress Inducers Enhances Oncolytic Adenovirus Enadenotucirev Replication and Killing through PKCα Activation: Molecular Therapy - Oncolytics

EGF-SubA enhances cytotoxic effects of the ER stress inducer... | Download  Scientific Diagram
EGF-SubA enhances cytotoxic effects of the ER stress inducer... | Download Scientific Diagram

HRC promotes anoikis resistance and metastasis by suppressing endoplasmic  reticulum stress in hepatocellular carcinoma
HRC promotes anoikis resistance and metastasis by suppressing endoplasmic reticulum stress in hepatocellular carcinoma

Endoplasmic reticulum stress signalling – from basic mechanisms to clinical  applications - Almanza - 2019 - The FEBS Journal - Wiley Online Library
Endoplasmic reticulum stress signalling – from basic mechanisms to clinical applications - Almanza - 2019 - The FEBS Journal - Wiley Online Library

Endoplasmic Reticulum Stress Mediates Vascular Smooth Muscle Cell  Calcification via Increased Release of Grp78 (Glucose-Regulated Protein, 78  kDa)-Loaded Extracellular Vesicles | Arteriosclerosis, Thrombosis, and  Vascular Biology
Endoplasmic Reticulum Stress Mediates Vascular Smooth Muscle Cell Calcification via Increased Release of Grp78 (Glucose-Regulated Protein, 78 kDa)-Loaded Extracellular Vesicles | Arteriosclerosis, Thrombosis, and Vascular Biology

Biomedicines | Free Full-Text | The Crosstalk of Endoplasmic Reticulum (ER)  Stress Pathways with NF-κB: Complex Mechanisms Relevant for Cancer,  Inflammation and Infection
Biomedicines | Free Full-Text | The Crosstalk of Endoplasmic Reticulum (ER) Stress Pathways with NF-κB: Complex Mechanisms Relevant for Cancer, Inflammation and Infection

Cell death induced by the ER stressor thapsigargin involves death receptor  5, a non-autophagic function of MAP1LC3B, and distinct contributions from  unfolded protein response components | Cell Communication and Signaling |  Full Text
Cell death induced by the ER stressor thapsigargin involves death receptor 5, a non-autophagic function of MAP1LC3B, and distinct contributions from unfolded protein response components | Cell Communication and Signaling | Full Text

Thapsigargin - Wikipedia
Thapsigargin - Wikipedia

Unfolded Protein Response (UPR) and Endoplasmic Reticulum (ER) Stress FAQs
Unfolded Protein Response (UPR) and Endoplasmic Reticulum (ER) Stress FAQs

Tunicamycin specifically aggravates ER stress and overcomes chemoresistance  in multidrug-resistant gastric cancer cells by inhibiting N-glycosylation |  Journal of Experimental & Clinical Cancer Research | Full Text
Tunicamycin specifically aggravates ER stress and overcomes chemoresistance in multidrug-resistant gastric cancer cells by inhibiting N-glycosylation | Journal of Experimental & Clinical Cancer Research | Full Text

What is Thapsigargin? Frequently Asked Questions
What is Thapsigargin? Frequently Asked Questions

Differential impact of imipramine on thapsigargin- and tunicamycin-induced endoplasmic  reticulum stress and mitochondrial dysfunction in neuroblastoma SH-SY5Y  cells - ScienceDirect
Differential impact of imipramine on thapsigargin- and tunicamycin-induced endoplasmic reticulum stress and mitochondrial dysfunction in neuroblastoma SH-SY5Y cells - ScienceDirect

Cell death induced by the ER stressor thapsigargin involves death receptor  5, a non-autophagic function of MAP1LC3B, and distinct contributions from  unfolded protein response components | Cell Communication and Signaling |  Full Text
Cell death induced by the ER stressor thapsigargin involves death receptor 5, a non-autophagic function of MAP1LC3B, and distinct contributions from unfolded protein response components | Cell Communication and Signaling | Full Text